PVC-induced cardiomyopathy resolves within 4 months of successful ablation in most patients. In multivariate analysis, only an epicardial PVC origin was predictive of delayed recovery of LV function in patients with PVC-induced cardiomyopathy. The PVC-QRS width was significantly longer in patients with delayed recovery than in patients with recovery within 4 months (170☒1 ms vs 159☑6 ms P =. An epicardial origin of PVCs was more often present (13 of 24, 54%) in patients with delayed recovery of LV function than in patients with early recovery of LV function (2 of 51, 4% P<.0001). In 24 (32%) patients, recovery of LV function took more than 4 months (mean 12☙ months range 5-45 months). The majority of patients (51 of 75, 68%) with PVC-induced LV dysfunction had a recovery of LV function within 4 months. The ejection fraction normalized at a mean of 5☖ months postablation. If LV function did not normalize after 3-4 months, a repeat echocardiogram was performed every 3 months until there was normalization or stabilization of LV function. In these patients, echocardiography was repeated 3-4 months postablation. The PVC burden was reduced to<20% of the initial PVC burden in 75 patients.
#Left ventricle ablation flutter series#
In a consecutive series of 264 patients with frequent idiopathic PVCs referred for PVC ablation, LV dysfunction was present in 87 patients (mean ejection fraction 40%☑0%). To describe the time course and predictors of recovery from LV dysfunction after effective ablation of PVCs in patients with PVC-induced cardiomyopathy. The time course of recovery of LV function has not been described. Patients with frequent premature ventricular complexes (PVCs) and PVC-induced cardiomyopathy usually have recovery of left ventricular (LV) dysfunction postablation.
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